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Does impaired processing of pro-B-type (or brain) natriuretic peptide cause decreased plasma BNP levels in obese heart failure patients?

   
@article{ATM28884,
	author = {Toshio Nishikimi and Yasuaki Nakagawa},
	title = {Does impaired processing of pro-B-type (or brain) natriuretic peptide cause decreased plasma BNP levels in obese heart failure patients?},
	journal = {Annals of Translational Medicine},
	volume = {7},
	number = {Suppl 6},
	year = {2019},
	keywords = {},
	abstract = {The natriuretic peptide system consists of three distinct endogenous peptides: atrial natriuretic peptide (ANP), brain (or B-type) natriuretic peptide (BNP), and C-type natriuretic peptide (CNP), as well as three receptors: natriuretic peptide receptor-A (NPR-A or guanynyl cyclase-A), natriuretic peptide receptor-B (NPR-B or guanynyl cyclase-B) and natriuretic peptide receptor-C (NPR-C or clearance receptor) (1). ANP and BNP are cardiac hormones, with ANP produced mainly in the atria and BNP produced mainly in the ventricles.},
	issn = {2305-5847},	url = {https://atm.amegroups.org/article/view/28884}
}